简介：Shuigou(GV26)isalsocalledRenzhong,locatinginthephiltrum,thejunctionofthesuperior1/3andthemiddle1/3.Itisoftenappliedtotreattheacutediseasesandsymptoms.Withregardtoacutelumbarsprainandacuterigidneck,Shuigou(GV26)functionstorelaxthemusclesandtendonsandbenefitthespinalcolumn.ItissaidinOdeoftheEssentialsofUnerstanding(TongXuanZhiYaoFu):'Shuigou(GV26)relievesthestiffnessandpainofthespinalcolumnanderectormuscleofspine.'ItissaidinSongsoftheJadeDragon(YuLongFu):'Forstiffnessandpainofspineandback,

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简介：Cancerisaleadingcauseofdeathworldwide.Cancertreatmentsbychemotherapeuticagents,surgery,andradiationhavenotbeenhighlyeffectiveinreducingtheincidenceofcancersandincreasingthesurvivalrateofcancerpatients.Inrecentyears,plant-derivedcompoundshaveattractedconsiderableattentionasalternativecancerremediesforenhancingcancerpreventionandtreatmentbecauseoftheirlowtoxicities,lowcosts,andlowsideeffects.Ellagicacid(EA)isanaturalphenolicconstituent.RecentinvitroandinvivoexperimentshaverevealedthatEAelicitsanticarcinogeniceffectsbyinhibitingtumorcellproliferation,inducingapoptosis,breakingDNAbindingtocarcinogens,blockingvirusinfection,anddisturbinginflammation,angiogenesis,anddrug-resistanceprocessesrequiredfortumorgrowthandmetastasis.ThisreviewenumeratestheanticarcinogenicactionsandmechanismsofEA.ItalsodiscussesfuturedirectionsontheapplicationsofEA.

简介：在男荷尔蒙代替治疗Finkelstein等。显示出那睾丸激素而非合成“；pure”；雄激素应该被规定。睾丸激素被变换成superactive雄激素dihydrotestosterone并且到estradiol，并且因此经由雄激素受体和两雌激素受体有行动(ERα；，ERβ；)。尽管肌肉力量是雄激素依赖者，estradiol在men—有主要生理的效果；在骨头上，软骨，并且和雄激素，在性工作上。既不dihydrotestosterone也不‘；pure’；合成雄激素能被变换成estradiol；那么对待的那些因此冒险错过有益(并且必要)在人的雌激素的效果。

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简介：Thepresentstudywasdesignedtoexaminetheanti-hyperuricemicandanti-inflammatoryeffectsandpossiblemechanismsofvaticaffinol,aresveratroltetramerisolatedfromethanolextractsofDipterocarpusalatus,inoxonate-inducedhyperuricemicmice.At1hafter250mg·kg~(-1)potassiumoxonatewasgiven,vaticaffinolat20,40,and60mg·kg~(-1)wasintragastricallyadministeredtohyperuricemicmiceoncedailyforsevenconsecutivedays.Vaticaffinolsignificantlydecreasedserumuricacidlevelsandimprovedkidneyfunctioninhyperuricemicmice.Itinhibitedhepaticactivityofxanthinedehydrogenase(XDH)andxanthineoxidase(XOD),regulatedrenalmRNAandproteinlevelsofuratetransporter1(URAT1),glucosetransporter9(GLUT9),organicaniontransporter1(OAT1),organiccationtransporter1(OCT1),OCT2,organiccation/carnitinetransporter1(OCTN1),andOCTN2inhyperuricemicmice.Moreover,vaticaffinolmarkedlydown-regulatedrenalproteinlevelsofNOD-likereceptor3(NLRP3),apoptosis-associatedspeck-like(ASC),andCaspase-1,resultinginthereductionofinterleukin(IL)-1β,IL-18,IL-6andtumornecrosisfactor-α(TNF-α)levelsinthisanimalmodel.Additionally,HPLCandLC-MSanalysesclearlytestifiedthepresenceofvaticaffinolinthecrudeextract.Theseresultssuggestthatvaticaffinolmaybeusefulforthepreventionandtreatmentofhyperuricemiawithkidneyinflammation.