简介：Objective:Toobservetheocularhistopathologicalchangesaftereyeballenucleationinducedbycornealtrauma.Methods:Lightmicroscopicexaminationwasdoneon117eyeballspecimensenucleatedaftercornealtrauma(18withcornealfissureand99withcornealperforatingtrauma).Results:Acute,subacuteorchronicinflammatorychanges,andfibrousmembraneformationwereobservedinwell-closedcornealwounds,whereasinflammation,atrophyandscarwereobservedinthefocaltissues.Butatthelateperiod,secondaryglaucoma,retinaldetachment,endophthalmitisandeyeballatrophyresultedinblindness.Cornealfistulawasobservedinthosewithinadequatecureofwoundscausedbyingrowthofcornealepithelium,embedmentofirisandvitreousbody,andlargeareaofcentrallylocatedtissuedeficiencyofthecorneal.Ahighincidenceofendophthalmitiswasnotedduetothepresenceofcornealfistula.Severeinflammationwasobservedintheanteriorsegmentaltissueswithfibrousinfiltrationintheanteriorchamber,whichmightresultinrapiddestructionoftheeyeballs.Conclusions:Ocularpathologyvarieswiththedifferenceoftheposition,form,sizeandclosingconditionsofthecorneallacerationaftertrauma.

简介：Objective:Tostudytheclinicaltherapeuticeffectofanisodamineonrespiratoryfunctionafterseverebraininjury.Methods:Ninetypatientswithrespiratorydysfunctionfollowingseverebraininjuryweredividedintotwogroups:atreatmentgroup(n=45,treatedwithroutinetherapyplusanisodamine)andacontrolgroup(n=45,treatedwithroutinetherapyonly).Thepulmonaryventilationfunctionandoxygenationfunctionwerecomparedbetweenthetwogroups.Results:Inthetreatmentgroup,12hoursaftertreatmenttherespiratoryratereduced,thepartialpressureofcarbondioxide(PCO2),thepartialpressureofoxygeninarterialblood(PaO2)andoxygenationexponentincreased,thedeadspaceventilationdoseandthepulmonaryalveolus-partialpressureofarterialoxygendifferencedecreased,andtheventilationfunctionoftherespiratorytractandpulmonaryoxygenationfunctionimproved.Therewasasignificantdifferencebetweenthetwogroups(P<0.01).Noside-effectwasfoundexceptaslightincreaseofintracranialpressureandheartrate.Conclusions:Anisodaminecanimprovepulmonaryventilationfunctionandoxygenationfunctionanddecreasetheincidenceofhypoxemiamarkedly.Itiseffectiveintreatingrespiratorydysfunctionafterseverebraininjury.

简介：ObjectiveToprovethehypothesisthatthereareassociationsbetweenbodyweightandinsulinresistanceinessentialhypertension,X-syndromesandtype-2diabetes.MethodsWeexaminedthebodymassindex(BMI)andinsulinsensitveindex(ISI)among70hypertensives,39X-syndromes,33type-2diabetesand46controls.ToobservetherelationsbetweenthedegreesofobesityandLSIinallstudygroups.Seventypatientswithhypertension[70hypertensives,48menand22women,meanage(69.3+6.7)years],39X-syndromes[27menand12women,meanage(66.4+6.2)years],33type-2diabetes[21menand12women,meanage(68.7±7.3)years],and46healthycontrols[36menand10women,meanage(66.1+6.6)years].Duringstudyperiod,nopatientsdied.ResultsAllpatientsofhypertensives,X-syndromesandtype-2diabeteshaveinsulinresistance,BMIinallpatientswassingnificantlyhigherthanincontrols.Thelevelsofbloodglucoseandinsulinweresignificantlyhigherinobesehypertensive

简介：Toprovidetheoreticalbasisforeffectandmechanismofpercutaneouslumbardiscectomyinclinic.Methods:Atotalof180patientswithlumbarintervertebraldischerniationwereevaluatedbyCTonthefifthdaybeforeandafteroperation.Meanwhile,CTvaluewasmeasuredinthedeterminedlevelandregion.Results:Afteroperation,CTvalueofthecentralandposteriordeterminedpointofherniatedintervertebraldiscwaslowersignificantlythanthatbeforeoperation(P<0.01),butCTvalueoftheanteriordeterminedpointwasdifferentinsignificantly.Theexcellentandgoodresultsofthepatientstogetherwere83%postoperatively.Conclusions:Thecurativeeffectofpercutaneouslumbardiscectomyisachievedthroughreductionoflumbarintradiscalpressure.

简介：Objective:Tosearchfortheinterferenceoppoaunityoffatemboliafterfractureoflongboneorpelvis.Methods:86patientswereselectedanddividedintourineketonepositivegroup(UKPgroup)andurineketonenegativegroup(UKNgroup).Theclinicaldata(includingclinicalandsub-clinicalfatembolism)ofallcaseswereanalyzed.Theincidencerateoffatembo-lismandtheserumtriglyceride(TG)levelwerecomparedbetweenthetwogroups.Results:7patientswithketonuriahadfatem-bolism,3ofthemhadprogressivebloodsedimentationandplateletabnormality.TheincidencerateoffatembolismandtheserumTGlevelinUKPgroupwashigherthanthatinUKNgroup.TherewasasignificantdifferencebetweentheUKPgroup(P<0.05)andtheUKNgroup(P<0.01).Conclusion:TheresultssuggestedthatthepatientshaveahigherriskoffatembolismwhentheserumTGlevelishigh,andresultsinketonuria.Sothiscanbeconsideredastheinterferenceopportunityofthefatemboli.

简介：Objective:Tostudytheeffectofhyperventilationonbraintissueoxygenpressure(PtiO2),braintissuecarbondioxidepressure(PtiCO2),pHvalueandintracranialpressure(ICP)duringintracranialhypertensioninpigs.Methods:Autologousarterialblood(5.5ml±0．5ml)wasinjectedintotheleftfrontallobebymicropumptoestablishthemodelofintracerebralhematomainpigs.Afterbloodinjection,theanimalswerehyperventilatedfor15minutestodecreasethepressureofcarbondioxideinarterialblood(PaCO2)to27.35mmHg±11.97mmHg(1mmHg=0.133kPa).Themeanarterialpressure(MAP),intracranialpressure(ICP),cerebralperfusionpressure(CPP),PtiO2,PtiCO2,pHvalueand[HCO3^-]werecontinuouslymonitoredandthebloodgaswasanalyzed.Results:Afterhyperventilation,theICPsignificantlydecreased(P<0.01),theCPPsignificantlyincreased(P<0.05),whilethePtiO2greatlydecreasedtotheischemiclevel(8.20mmHg±2.50mmHg)(P<0.01),thePtiCO2decreased(P<0.01)andthepHvalueincreased(P<0.01).Atthesametime,bloodgasanalysisshowedthatthePaCO2greatlydecreasedandthepHvalueincreased.Conclusions:HyperventilationcandecreasetheICPandthePtiO2significantly.Therefore,hyperventilationshouldbeavoidedearlyafterbraininjury.ThePtiO2monitoringwillbehelpfulfordetectingcerebralischemiaearly.

简介：ObjectiveTo观察激活p21的kinase6(PAK6)在在成年rat.MethodsSprague-Dawley老鼠的针的绳索损害(SCI)以后的表示和它的可能的角色受到针的绳索损害。为了探索PAK6，表示模式和PAK6的分发的病理学、生理的意义，没被西方的污点观察，免疫组织化学和immunofluorescence.ResultsWestern污点分析证明PAK6蛋白质水平在白天2和白天4上是显著地起来调整的，然后减少了并且有到白天14为止的起来规定。免疫组织化学分析证明PAK6的表示显著地在与控制组相比的白天4上被增加。而且，染色的两倍immunofluorescence证明PAK6首先在控制组在神经原和星形细胞被表示。当时在损害以后，PAK6的表示在星形细胞显著地被增加，神经原，和星形细胞大部分被增殖。我们也检验了增殖的房间的表示原子抗原(PCNA)并且发现它的变化与PAK6的表示被相关。重要地，在在受伤针的绳索的PAK6的injury.ConclusionThe起来规定可以与glial增长被联系以后，染色的两倍immunofluorescence表明PCNA评估的房间增长在白天4上出现在许多PAK6-express-ing房间。

简介：Objective:Todeterminetheefficacyofpolylacticacidglueinpreventingepiduralscaradhesionafterlaminectomyinrabbits.Methods:Twenty-fourJapanesewhiterabbitsunderwentlaminectomy(includingtheattachedligaments)atL2andL5.AfterlaminectomyatL5,polylacticacidgluewassprayedontheduraandnerverootsandthissegmentwastakenastheexperimentalgroup.AfterlaminectomyatL2,nothingwasusedandthissegmentwasenrolledastheselfcontrolgroup.Fourrabbitswerekilledeverytwoweekspostoperativelytilltheendoftheexperimentat12weeks.Thentheoperatedspinewasobservedgrossly,histologicallyandultrastructurallytocheckthedegreeofscarformation,thestatusofepiduralscaradhesion,theabsorptionoftheglue,andtheintracellularstructureoffibroblasts.Results:Thegluecoagulatedimmediatelyaftersprayingandshowedexcellenthemostaticeffect.Thegluemembranewaseasytobetakenawayfromtheduramaterofthesamplesfor2weeksandtherewerenocellsintheepiduralspaceintheexperimentalgroup.Buttheduramaterwascoveredbyhematomainthecontrolgroup,whichformedmildadhesion,withfibroblastsproliferatingactively.Inthe4thweek,someglueshiversremainedintheepiduralspacewithfibroblastsincreasingalittle,andtheduramaterwassmoothintheexperimentalgroup.However,inthecontrolgroup,theformedscarwasfragileandconglutinatedwiththeduramaterdiffuselyandfibroblastsweremuchmorethanthoseintheexperimentalgroup.Inthe6th-12thweeks,therewasapotentialinterspacebetweenthescarandtheduramater,andthepolylacticacidgluewasabsorbedcompletelyintheexperimentalgroup.Muchtoughscarwasfoundinthecontrolgroup,whichwasverydifficulttodissectfromtheduramaterandthesurroundingtissues.Fromtheultrastructuralobservationofthefibroblasts,thenucleusbecamemuchbiggerandtheroughendoplasmicreticulumwasmuchmoreplentifulinthecontrolgroupthanthatinthee

简介：客观：没有主要肺的损害，在机械通风治疗以后与尖锐服的损害在病人的唾沫检测dipalmitoylphosphatidyl胆碱(DPPC)的层次。方法：在有尖锐服的损害，但是没有肺的损害的35个病人的唾沫的DPPC层次在通风和16-20的开始与高效液体层析被检测在通风以后的天，21-40天，和41-60天分别地。结果：没有在在通风以后的16-20天之间的DPPC层次的重要差别(3.36±0.49)并且在通风(3.37±0.58)的开始(P>0.05)。DPPC的Themean层次在21-40天显著地减少了(2.87mg/ml±0.26nig/ml，P<0.05)and41-60天(1.93mg/ml±0.21mg/ml，P<0.01)在通风以后与那相比在通风的开始。在一样的时期，吸入的压力和平均数航线迫使的山峰显著地增加了，而静态的依从和在动脉的氧的部分压力显著地减少了。在收到了通风超过20天的25个病人之中，(32%)8在通风的开始与那相比在动脉有氧的稍微减少的部分压力。结论：DPPC铺平的机械通风罐头减少，减少肺依从和增加航线压力，甚至与尖锐服的损害损害氧化功能住院病人。反常DPPC是主要原因之一联系ofventilator的肺损害。

简介：

简介：Toexplorethemolecularmechanismoftheprotectiveeffectofnervegrowthfactor(NGF)oninjuredspinalcord.Methods:TheposteriorT8(the8ththoracicsegment)spinalcordsof60Wistarratswereinjuredbyimpactscausedbyobjects(weighing10g)fallingfromaheightof2.5cmwithAllensway.Solutionwithnervegrowthfactors(NGF)wasgivento30rats(theNGFgroup)throughamicrotubuleinsertedintothesubarachnoidcavityimmediately,andat2,4,8,12and24hoursafterspinalcordinjury(SCI)respectively.Normalsaline(NS)withsamevolumewasgiventotheother30rats(theNSgroup)withthesamemethod.And5normalratsweretakenasthenormalcontrols.Theexpressionofbcl-2andbaxproteinsinspinalcordwasdetectedwithimmunohistochemistry.Theapoptoticneuronsinspinalcordweremeasuredwithterminaldeoxynucleotidyltransferase-mediateddUTP-biotinnickend-labelingofDNAfragments(TUNEL)staining.Results:Thepositiveexpressionofbcl-2proteinwasstronginthenormalcontrols,butdecreasedintheNSgroup,andincreasedsignificantlyintheNGFgroupascomparedwiththatoftheNSgroup(P<0.01).Thepositiveexpressionofbaxproteinwasalsostronginthenormalcontrols,butincreasedintheNSgroup,anddecreasedsignificantlyintheNGFgroupascomparedwiththatoftheNSgroup(P<0.01).ApoptoticneuronswerefoundintheNSgroup,andtheydecreasedsignificantlyintheNGFgroupascomparedwiththatoftheNSgroup(P<0.01).Conclusions:NGFcanprotecttheinjurednervetissuesthroughstimulatingtheexpressionofbcl-2protein,inhibitingtheexpressionofbaxproteinandinhibitingtheneuronalapoptosisafterSCI.

简介：Objective:　Toexplorethevariantprocessesofcellapoptosisandtheinhibitingeffectofmoderatehypothermiaoncellapoptosisafterdiffusebraininjury.　　Methods:　ModelsofdiffusebraininjurywereinducedbythetraumadevicereportedbyMarmarou.1Atotalof128Wistarratsweredividedinto4groups:theuninjuredgroup(GroupA,n=8),theseverelyinjuredgroup(GroupB,n=60),themildlyinjuredgroup(GroupC,n=30)andthemildhypothermiagroup(GroupD,n=30).InGroupD,theseverelyinjuredratsweretreatedwithmoderatehypothermiatokeeptherectaltemperatureat32℃(standarddeviationfor0.1℃)for6hours.Thenthemorphosis,thecharacteristicsandthequantityofapoptoticcellsinthecerebralcortexandinthehippocampusregionsafterdifferentseveritiesofcraniocerebralinjurieswereobservedandcomparedunderanelectronicmicroscope,withterminaldeoxynucleotidylnickendlabeling(TUNEL)inDNAfragmentationandwithagarosegelelectrophoresis.　　Results:　TUNELshowedapoptoticcellsincreasedaccordingtotheinjuryseverity,andtheypeakedat48hoursafterinjuryandthendeclined.InGroupC,apoptosiswaslocatedintheCA2andCA3areasofthehippocampus.AndinGroupB,apoptosisincreasedevidently,andlocatedinthewholehippocampusandinthefrontalandparietalcortexregions.Thehypothermia-treatedratshadsomeapoptoticcells,too.However,evenat24,48and72hoursafterinjurythereweresignificantlyfewerapoptoticcellsinthecortexandinthehippocampusinGroupDthanthatinthenon-treatedgroups.Electronmicroscopyshowedthattheapoptoticcellswereroundandshrunkeninmorphologyandthenucleiwereroundandcondensedat24and48hoursafterinjury.Andtheapoptosisat48hourswasmoreseverethanthatat24hours.Thehypothermia-treatedratshadnoapoptoticcells.GelelectrophoresisshowedthatcharacteristicDNA“ladders”wereobservedinthecortexandinthehippocampusat48hoursafters

简介：Objective:Tobetterunderstandthecharacteristicsoftheneurogenicmotorevokedpotential(NMEP)beforeandafteracutespinalcordinjury.Methods:WerecordedandcharacterizedthespinalcordNMEPfrom48normalratsandfrom38ratswithspinalcordhemisectionlesion.SpinalcordNMEPswereelicitedbyapplyingarangeofcurrentintensitieswithbipolarmicroelectrodestimulitotheC4cordsegmentandrecordingtheresponsesfromsciaticnerveswithbipolarmicroelectrodesplacedintheneurilemma.Results:Theevokedpotentialsconsistedofthreestableandreproduciblenegativeandthreepositivepeaks.Themean±SDlatenciesofN1were2.89±0.22msontherightsideand2.89±0.24msontheleftside.Themeanconductionvelocitywas47.9m/s.Themean±SDamplitudesofN1were3.61±2.10μVontherightsideand3.83±2.32μVontheleftside.TheamplitudesofN1weresignificantlydifferentamongtheeightstimulusintensitygroups(rightside:F=2.22,df=7201,P=0.03;leftside:f=2.11,df=7206,P=0.04).Theamplitudewaslargestwhenthestimulusintensitywas1.1-2.5mA.ThelatenciesofN1werenotsignificantlydifferentamongtheeightstimulusintensitygroups(rightside:F=0.40,df=7201,P=0.9;leftside:F=1.20,df=7206,P=0.3).TheamplitudesandlatenciesofN2,N3werenotsignificantlydifferentamongtheeightstimulusintensitygroups.Therewerenosignificantchangesinlatencyandamplitudebetweentheleftandtherightsidenerveresponses.Thirty-eightratsundewentT9cordrightsidehemisection.Amongthem,20(53%),30(79%),and32(84%)ratscouldnotberecordedincorrespondingtoN1,N2,andN3,respectively,intheright-sidesciaticnerves;and13(79%),18(47%),and21(55%),incorrespondingtoN1,N2,andN3intheleft-sidesciaticnerves.Thelatencywassignificantlydelayedonthebothrightandleftsides.TheamplitudeN1wassignificantlydepressedonthebothsides,withN3significantlydepressedontherightsideandN2notsignificantlydepressed.Conclusions:TheamplitudeofNissignificantlydiffere

简介：

简介：Objective:ToinvestigateCaspase-3expressionanditsroleinneuronalapoptosis.Methods:TheT13-L2spinalcordofratswasinjuredbytractionaftertheamplitudeofP1-N1wave,monitoredbyacorticalsomatosensoryevokedpotential(CSEP)monitor,decreasedtoseventypercentofthatbeforeoperation.Thenratswerekilledin6h,1d,4d,7d,14dand21drespectivelyafteroperation.Flowcytometerterminaldeoxynucleotldyltransferease-mediatedbiotinylateddeoxynuridinetriphosphatenickendlabeling(TUNEL),Caspase-3activityassayandimmunohistochemicalmethodwereappliedtoinvestigateCaspase-3expressioninthespinalcordtissueandtostudyneuronalapoptosisinrats.Results:Afterspinalcordinjury,apoptoticcellsdetectedbyflowcytometryandTUNEL-positivecellsweresignificantlymore,andpositiveimmunohistochemicalstainingofCaspase-3andCaspase-3activityweresignificantlyhigherinGroupinjurythaninGroupscontrolandlaminectomy,respectively(P>0.05,P>0.01).Similartrendofchangeswasnoticedinapoptoticcells,TUNEL-positivecellsandpositiveimmunohistochemicalstainingofCaspase-3,allofwhichreachedtheirrespectivepeak7daysafteroperation.Caspase-3activityreacheditspeak,however,4dayspostoperatively.Conclusions:IncreasedexpressionandactivityofCaspase-3proteininneuronsaftertractivespinalcordinjuryisthebiochemicalsignalofearlyspinalcellapoptosis.Itisofgreatsignificanceforunderstandingthemechanismofspinalcordinjury.

简介：Objective:TostudythechangesofthegeneexpressionpatternofspinalcordtissuesintheearlystageafterinjurybyDNAmicroarray(genechip).Methods:ThecontusionmodelofratspinalcordwasestablishedaccordingtoAllen'sfallingstrikemethodandthegeneexpressionpatternsofnormalandinjuredspinalcordtissueswerestudiedbygenechip.Results:Theexpressionof45geneswassignificantlychangedintheearlystageafterspinalcordinjury,inwhich22genesup-regulatedand23genesdown-regulated.Conclusions:Theexpressionofsomegeneschangessignificantlyintheearlystageafterspinalcordinjury,whichindicatesthecomplexityofsecondaryspinalcordinjury.

简介：Objective:Tocomparativelystudythedifferenteffectsofopenheartsurgeryonbraintissuesofpatientswithcongenitalandrheumaticheartdisease.Methods:Fortypatientswithcongenitalheartdisease(CHD,CHDgroup,n=20)orrheumaticheartdisease(RHD,RHDgroup,n=20)underwenton-pump(cardiopulmonarybypass,CPB)heart-beatingopenheartsurgery.BloodsamplesbeforeCPB,and20minutes,1hour,24hoursand7daysafterCPBwerecollected,andthelevelsofneuron-specificenolase(NSE)andproteinS-100bintheplasmaweredeterminedwithenzyme-linkedimmunoadsorbentassay(ELISA),respectively.Allthepatientswereexaminedwithelectroencephalogram(EEG)beforeand1weekafteroperation.ThechangesofNSE,S-100bandEEGcomparedtoverifythedifferenceofpostoperativecerebralinjurybetweenCHDcasesandRHDcases.Results:TheplasmalevelofS-100bincreasedsignificantly20minutesafterCPBandwasstillhigherthanthepreoperativelevelat24hoursafteroperationinbothgroups(P<0.01).TheplasmalevelofNSEincreasedmoresignificantlyintheCHDgroupthanintheRHDgroup20minutesafterCPBanditreturnedtothenormallevel24hoursafterCPBintheCHDgroupbutremainedatahighlevelintheRHDgroup(P<0.01).ThelevelsofNSEandS-100breturnedtothenormallevelsonthe7thdayafterCPB.AbnormalEEGwasfoundin75%ofthepatientsintheCHDgroupand60%intheRHDgroup.Conclusions:On-pumpheart-beatingopenheartsurgerycancausecertaincerebralinjuryinthepatientswithCHDorRHD.TheinjurywasmoresevereandrecoveredmorequicklyintheCHDgroupthanintheRHDgroup.

简介：客观：在兔子在针的绳索ischemia/reperfusion(I/R)以后在lipidperoxidation和apoptosis上学习白果树biloba摘录(GBE)的效果。方法：SpinalcordI/R损害模型根据对Erten等的描述被建立。27只NewZealand白兔子的一个总数随机被划分成三个组：一个假冒的组(9只兔子对待withs火腿操作但是没有大动脉的吸藏)，一个模型组(与大动脉的吸藏对待的9只兔子并且匹配卷盐)，并且一个GBE组(与大动脉的吸藏andGinaton(100mg/kg)对待的9只兔子在大动脉的夹钳前并且在灌注的发作注射了30分钟)。Theneurological结果分别地在灌注以后在24和48个小时被评估。针的绳索malondialdehyde(MDA)水平，超级氧化物dismutase(草皮)然后被检测。神经cellapoptosis被终端deoxynucleotidyltransferase(TdT)决定标记的-mediateddUTP-fluorescence刻痕结束(TUNEL)方法和bcl-2和bax的表示是examinedhistologically在有免疫组织化学的针的绳索。结果：I/Rproduced在神经病学的得分的重要减少。GBE组的马达分数比在在灌注以后的24和48个小时的模型组的那些显著地高(P<0.05)。与模型组相比，GBE改善了草皮的下面规定并且生产了MDA水平的重要减小(P<0.01)。为在模型组的TUNEL的积极房间是多于GBE组的那些的大部分(P<0.01)。bcl-2在I/R以后是起来调整的，特别在theGBE组(P<0.01)。bax的起来规定被GBE极大地减少(P<0.01).Conclusions：GBE对针的绳索I/R损害，和机制有保护的效果可以是它能清除氧释放激进分子并且禁止神经房间的apoptosis。

简介：Objective:Toexplorethecausesoftheformationoftraumaticcarotid-cavernousfistulasandthetherapeuticeffectofdetachableballoonand/orcoilembolizationandthepreventionofitscomplications.Methods:FromOctober,1992toMarch,2002,17patientswithtraumaticcarotid-cavernousfistulasweretreatedwithdetachableballoonand/orcoilembolizationinourhospital.TheclinicaldataandimagingfeaturesofCT,MRandselectiveangiogramofthesepatientswereanalyzed.Results:Oneweekaftertreatmentwithembolization,theclinicalsymptomsofthe17patientswereremitted,andopticcacophony,nystagmus,exophthalmosanddropsyofconjunctivadisappeared.Twopatientsmanifestedsurgicalcomplications,onepatientdied.Sixteenpatientssurvived.Theywereallfollowedupformorethan2years,whichshowedonepatienthadhandicapinmovement,andinonepatientthesignsandsymptomsoftraumaticcarotid-cavernousfistulasreoccurred2monthsaftertreatment.Conclusions:Thedetachableballoonand/orcoilembolizationissafeandreliable.Itisagoodmethodtotreattraumaticcarotid-cavernousfistulas.

简介：ObjectiveThebrainTXA_2andPGl_2levelsinanewrodentmodelofimpactaccelerationdiffusebraininjuryaloneandwithhypotentionandhyperthermiaincombinationwereob-servedtolookintotherelationshipbetweenTXA_2,PGI_2levelsanddifferenttypesofheadinjury.MethodsThirty-twoSDratswereran-